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Damaged axillary nerve
Damaged axillary nerve


Axillary nerve dysfunction

Definition:

Axillary nerve dysfunction is a loss of movement or sensation of the shoulder because of damage to the axillary nerve.



Alternative Names:
Neuropathy - axillary nerve

Causes, incidence, and risk factors:

Axillary nerve dysfunction is a form of peripheral neuropathy. It occurs when there is damage to the axillary nerve, which supplies the deltoid muscles of the shoulder. It is not a specific, separate disease, but is caused by many conditions that can damage the axillary nerve.

Dysfunction of a single nerve group, such as the axillary nerve, is called mononeuropathy. Mononeuropathy implies a local cause of the nerve damage, although systemic disorders may occasionally cause isolated nerve damage (such as occurs with mononeuritis multiplex).

The usual causes are direct trauma, prolonged external pressure on the nerve, and pressure on the nerve from nearby body structures.

Conditions associated with axillary nerve dysfunction include fracture of the humerus (upper arm bone), pressure from casts or splints, improper use of crutches, intramuscular injections, and shoulder dislocation. Other causes include systemic disorders that cause neuritis (inflammation of nerves).

In some cases, no detectable cause can be identified. Mechanical factors may be complicated by ischemia (a lack of oxygen from decreased blood flow) in the area.




Review Date: 2/7/2005
Reviewed By: Luc Jasmin, MD, PhD, Departments of Anatomy & Neurological Surgery, University of California, San Francisco, CA. Review provided by VeriMed Healthcare Network.

The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. A licensed physician should be consulted for diagnosis and treatment of any and all medical conditions. Call 911 for all medical emergencies. Links to other sites are provided for information only -- they do not constitute endorsements of those other sites. Copyright 2004 A.D.A.M., Inc. Any duplication or distribution of the information contained herein is strictly prohibited.

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